Clinical Presentation - Febrile Non-Hemolytic Transfusion Reactions

Signs and symptoms of a Febrile Non-Hemolytic Transfusion Reaction (FNHTR) can appear during the transfusion or within four hours of its completion. They may include:

  • fever (>/= 38 degrees C and a change of  >/= 1 degree C from pretransfusion value)
  • chills
  • sensation of cold or
  • rigors
  • headache
  • nausea

Mechanisms

FNHTR Mediated by Antibodies

The proposed mechanism for FNHTR to RBC is an antigen-antibody reaction between recipient antibodies to antigens on donor leukocytes in which cytokines are released by donor leukocytes. The cytokines released include interleukin 1 b (IL-1 b), interleukin 6 (IL-6), and tumor necrosis factor (TNF).

Another theory is that the recipient-donor WBC antigen-antibody reaction may activate complement, thus stimulating the recipient's macrophages to produce and release cytokines.

FNHTR Mediated by Accumulation of Cytokines During Storage

According to one proposed mechanism for FNHTR associated with transfusion of platelets, cytokines (IL-1 b,, IL-6, TNF), and possibly other biological response mediators, accumulate in the plasma in which platelets are stored and, when infused, cause the symptoms typical of these reactions. However, prestorage leukoreduction has not entirely eliminated the reactions, as would be expected if leukocyte-derived cytokines were solely responsible.

Because many other biologic response modifiers, including platelet-derived cytokines, chemokines, complement fragments, histamine, and lipids, also accumulate in platelets during storage, and it is possible that these may play a role in the residual reactions. The possibly multifactorial mechanisms of the residual reactions require further investigation. 
 

Events Calendar

S M T W T F S
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 

Text will be replaced with image replacement